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Overexpression of the Flii gene increases dermal-epidermal blistering in an autoimmune ColVII mouse model of epidermolysis bullosa acquisita

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dc.contributor.authorKopecki, Zlatko
dc.contributor.authorArkell, Ruth
dc.contributor.authorStrudwick, Xanthe L
dc.contributor.authorHirose, Misa
dc.contributor.authorLudwig, Ralf J.
dc.contributor.authorKern, Johannes S.
dc.contributor.authorBruckner-Tuderman, Leena
dc.contributor.authorZillikens, Detlef
dc.contributor.authorMurrell, Dedee F.
dc.contributor.authorCowin, Allison J
dc.date.accessioned2015-12-10T23:12:45Z
dc.date.issued2011
dc.date.updated2016-02-24T12:09:11Z
dc.description.abstractEpidermolysis bullosa (EB) is a severe genetic skin fragility syndrome characterized by blister formation. The molecular basis of EB is still largely unknown and wound healing in patients suffering from EB remains a major challenge to their survival. Our
dc.identifier.issn0022-3417
dc.identifier.urihttp://hdl.handle.net/1885/64116
dc.publisherJohn Wiley & Sons Inc
dc.sourceJournal of Pathology
dc.subjectKeywords: actin; alpha smooth muscle actin; collagen type 7; integrin; protein flightless 1; Smad2 protein; Smad3 protein; transforming growth factor beta1; unclassified drug; allele; animal cell; article; blister; cell adhesion; cell differentiation; cell prolifer epidermolysis bullosa; Flightless; Flii; TGF-ß; type VII collagen; wound healing
dc.titleOverexpression of the Flii gene increases dermal-epidermal blistering in an autoimmune ColVII mouse model of epidermolysis bullosa acquisita
dc.typeJournal article
local.bibliographicCitation.lastpage413
local.bibliographicCitation.startpage401
local.contributor.affiliationKopecki, Zlatko, Women's and Children's Health Research Institute
local.contributor.affiliationArkell, Ruth, College of Medicine, Biology and Environment, ANU
local.contributor.affiliationStrudwick, Xanthe L, Women's and Children's Health Research Institute
local.contributor.affiliationHirose, Misa, University of Lubeck
local.contributor.affiliationLudwig, Ralf J., University of Lubeck
local.contributor.affiliationKern, Johannes S., Germany and Freiburg Institute for Advanced Studies
local.contributor.affiliationBruckner-Tuderman, Leena, Germany and Freiburg Institute for Advanced Studies
local.contributor.affiliationZillikens, Detlef, University of Lubeck
local.contributor.affiliationMurrell, Dedee F., University of New South Wales
local.contributor.affiliationCowin, Allison J, Women's and Children's Health Research Institute
local.contributor.authoruidArkell, Ruth, u4350791
local.description.embargo2037-12-31
local.description.notesImported from ARIES
local.identifier.absfor060103 - Cell Development, Proliferation and Death
local.identifier.absfor060403 - Developmental Genetics (incl. Sex Determination)
local.identifier.absseo970106 - Expanding Knowledge in the Biological Sciences
local.identifier.ariespublicationu9511635xPUB889
local.identifier.citationvolume225
local.identifier.doi10.1002/path.2973
local.identifier.scopusID2-s2.0-80054719489
local.identifier.thomsonID000296369900010
local.type.statusPublished Version

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