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Overexpression of the Flii gene increases dermal-epidermal blistering in an autoimmune ColVII mouse model of epidermolysis bullosa acquisita

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Date

2011

Authors

Kopecki, Zlatko
Arkell, Ruth
Strudwick, Xanthe L
Hirose, Misa
Ludwig, Ralf J.
Kern, Johannes S.
Bruckner-Tuderman, Leena
Zillikens, Detlef
Murrell, Dedee F.
Cowin, Allison J

Journal Title

Journal ISSN

Volume Title

Publisher

John Wiley & Sons Inc

Abstract

Epidermolysis bullosa (EB) is a severe genetic skin fragility syndrome characterized by blister formation. The molecular basis of EB is still largely unknown and wound healing in patients suffering from EB remains a major challenge to their survival. Our

Description

Keywords

Keywords: actin; alpha smooth muscle actin; collagen type 7; integrin; protein flightless 1; Smad2 protein; Smad3 protein; transforming growth factor beta1; unclassified drug; allele; animal cell; article; blister; cell adhesion; cell differentiation; cell prolifer epidermolysis bullosa; Flightless; Flii; TGF-ß; type VII collagen; wound healing

Citation

URI

http://hdl.handle.net/1885/64116

Collections

ANU Research Publications

Source

Journal of Pathology

Type

Journal article

Book Title

Entity type

Access Statement

License Rights

DOI

10.1002/path.2973

Restricted until

2037-12-31

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Description
01_Kopecki_Overexpression_of_the__Flii__2011.pdf (1.81 MB)

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