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Hepatic ischemia reperfusion injury: Pathogenic mechanisms and basis for hepatoprotection

dc.contributor.authorTeoh, Narcissus
dc.contributor.authorFarrell, Geoff C
dc.date.accessioned2015-12-10T22:17:14Z
dc.date.issued2003
dc.date.updated2015-12-09T08:30:58Z
dc.description.abstractThis review highlights recent advances in our understanding of mechanisms underlying repeffusion injury to the liver after warm hepatic ischemia. Sinusoidal endothelial cells and hepatocytes are targets of injury in the early 'cytotoxic' phase, although p
dc.identifier.issn0815-9319
dc.identifier.urihttp://hdl.handle.net/1885/51310
dc.publisherBlackwell Publishing Ltd
dc.sourceJournal of Gastroenterology and Hepatology
dc.subjectKeywords: cell adhesion molecule; chemokine; cytokine; interleukin 1; interleukin 6; nitric oxide; reactive oxygen metabolite; tumor necrosis factor; tumor necrosis factor alpha; cell death; energy metabolism; human; human cell; hypoxia; inflammatory cell; Kupffer Adenosine; Adhesion molecules; Cell-cycle entry; Hepatic ischemia reperfusion injury; Ischemic preconditioning; Kupffer cells; Microcirculation; Neutrophils; Nitric oxide; Reactive oxygen species; Tumor necrosis factor
dc.titleHepatic ischemia reperfusion injury: Pathogenic mechanisms and basis for hepatoprotection
dc.typeJournal article
local.bibliographicCitation.lastpage902
local.bibliographicCitation.startpage891
local.contributor.affiliationTeoh, Narcissus, College of Medicine, Biology and Environment, ANU
local.contributor.affiliationFarrell, Geoff C, University of Sydney
local.contributor.authoruidTeoh, Narcissus, u4325419
local.description.embargo2037-12-31
local.description.notesImported from ARIES
local.identifier.absfor110307 - Gastroenterology and Hepatology
local.identifier.ariespublicationu4222028xPUB221
local.identifier.citationvolume18
local.identifier.doi10.1046/j.1440-1746.2003.03056.x
local.identifier.scopusID2-s2.0-0042855206
local.type.statusPublished Version

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