Cultural advice

The Australian National University acknowledges, celebrates and pays our respects to the Ngunnawal and Ngambri people of the Canberra region and to all First Nations Australians on whose traditional lands we meet and work, and whose cultures are among the oldest continuing cultures in human history.

Aboriginal and Torres Strait Islander peoples are advised that ANU Library collections may include images, names, voices, and other representations of deceased persons.

Material in the collection may contain terms, language or views that reflect the period in which the item was created and may be considered inappropriate today.

The NF-κB transcription factor c-Rel is required for Th17 effector cell development in experimental autoimmune encephalomyelitis

Thumbnail Image

Date

2011

Authors

Chen, Guobing
Hardy, Kristine
Pagler, Eloisa
Ma, Lina
Lee, Seung Soo
Gerondakis, Steve
Daley, Stephen
Shannon, M Frances

Journal Title

Journal ISSN

Volume Title

Publisher

American Association of Immunologists

Abstract

Experimental autoimmune encephalomyelitis (EAE) is a T cell-mediated autoimmune disease involving effector Th subsets such as Th1 and Th17. In this study, we demonstrate that mice lacking the NF-κB transcription factor family member c-Rel (rel -/-), whic

Description

Keywords

Keywords: immunoglobulin enhancer binding protein; interleukin 2; retinoid related orphan receptor gamma; T lymphocyte receptor; transcription factor Rel; allergic encephalomyelitis; animal cell; animal experiment; article; bone marrow; cell maturation; chimera; co

Citation

URI

http://hdl.handle.net/1885/52574

Collections

ANU Research Publications

Source

Journal of Immunology

Type

Journal article

Book Title

Entity type

Access Statement

License Rights

DOI

10.4049/jimmunol.1101757

Restricted until

2037-12-31

Downloads

File
Description
01_Chen_The_NF-κB_transcription_factor_2011.pdf (2.06 MB)

DSpace software copyright © 2002-2026 LYRASIS