Cultural advice

The Australian National University acknowledges, celebrates and pays our respects to the Ngunnawal and Ngambri people of the Canberra region and to all First Nations Australians on whose traditional lands we meet and work, and whose cultures are among the oldest continuing cultures in human history.

Aboriginal and Torres Strait Islander peoples are advised that ANU Library collections may include images, names, voices, and other representations of deceased persons.

Material in the collection may contain terms, language or views that reflect the period in which the item was created and may be considered inappropriate today.

IL-5 contributes to worm expulsion and muscle hypercontractility in a primary T. spiralis infection

Thumbnail Image

Date

1999

Authors

Vallence, Bruce
Blennerhasset, P
Deng, Y
Matthaei, Klaus
Young, I M
Collins, Stephen Frederick

Journal Title

Journal ISSN

Volume Title

Publisher

American Physiological Society

Abstract

Enteric nematode infections lead to increased interleukin (IL)-5 expression, eosinophilic inflammation, and intestinal smooth muscle hypercontractility. Although eosinophils release inflammatory mediators that cause smooth muscle contraction, the role of

Description

Keywords

Keywords: interleukin 5; animal experiment; animal model; animal tissue; article; controlled study; cytokine production; eosinophilia; inflammation; intestine infection; intestine muscle; male; mouse; nonhuman; priority journal; smooth muscle contraction; trichinel Eosinophils; Intestine; Nematode

Citation

URI

http://hdl.handle.net/1885/92468

Collections

ANU Research Publications

Source

American Journal of Physiology: Gastrointestinal and Liver Physiology

Type

Journal article

Book Title

Entity type

Access Statement

License Rights

DOI

Restricted until

DSpace software copyright © 2002-2026 LYRASIS