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Tudor griffith, gap junctions and conducted vasodilatation: Electromechanical coupling back in the limelight

dc.contributor.authorHill, Caryl
dc.date.accessioned2015-12-13T22:16:19Z
dc.date.issued2013
dc.date.updated2016-02-24T08:58:21Z
dc.description.abstractABSTRACT:: Tudor Griffith's untimely death cut short a research career focused on the mechanisms regulating vascular tone and blood flow. This brief review highlights the contribution that Tudor's work made to 3 main areas: the early days of study toward
dc.identifier.issn0160-2446
dc.identifier.urihttp://hdl.handle.net/1885/70804
dc.publisherLippincott Williams & Wilkins
dc.sourceJournal of Cardiovascular Pharmacology
dc.subjectKeywords: adenosine triphosphatase (potassium); calcium channel; connexin 43; cyclic AMP; endothelium derived hyperpolarizing factor; gap junction protein; nitric oxide; unclassified drug; voltage dependent calcium channel; article; blood vessel reactivity; calcium conducted vasodilatation; endothelium-derived hyperpolarization; gap junctions
dc.titleTudor griffith, gap junctions and conducted vasodilatation: Electromechanical coupling back in the limelight
dc.typeJournal article
local.bibliographicCitation.issue2
local.bibliographicCitation.lastpage101
local.bibliographicCitation.startpage93
local.contributor.affiliationHill, Caryl, College of Medicine, Biology and Environment, ANU
local.contributor.authoruidHill, Caryl, u8200545
local.description.embargo2037-12-31
local.description.notesImported from ARIES
local.identifier.absfor110200 - CARDIOVASCULAR MEDICINE AND HAEMATOLOGY
local.identifier.ariespublicationf5625xPUB2423
local.identifier.citationvolume61
local.identifier.doi10.1097/FJC.0b013e31827687c8
local.identifier.scopusID2-s2.0-84873408350
local.identifier.thomsonID000314807700002
local.type.statusPublished Version

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