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Increased permeability of the malaria-infected erythrocyte to organic cations

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dc.contributor.authorStaines, H
dc.contributor.authorRae, Caroline
dc.contributor.authorKirk, Kiaran
dc.date.accessioned2015-12-13T23:14:52Z
dc.date.issued2000
dc.date.updated2015-12-12T08:40:44Z
dc.description.abstractThe human malaria parasite, Plasmodium falciparum, induces in the plasma membrane of its host red blood cell new permeation pathways (NPP) that allow the influx of a variety of low molecular weight solutes. In this study we have demonstrated that the NPP
dc.identifier.issn0006-3002
dc.identifier.urihttp://hdl.handle.net/1885/88825
dc.publisherElsevier
dc.sourceBiochimica et Biophysica Acta: International journal of Biochemistry and Biophysics
dc.subjectKeywords: cation; choline; phenyltrimethylammonium; quaternary ammonium derivative; tetrabutylammonium; tetramethylammonium; tetrapentylammonium; tetrapropylammonium; tetrylammonium; article; controlled study; drug structure; erythrocyte; hemolysis; human; human ce Channel; Induced transport; Membrane permeability; Plasmodium falciparum; Selectivity
dc.titleIncreased permeability of the malaria-infected erythrocyte to organic cations
dc.typeJournal article
local.bibliographicCitation.lastpage98
local.bibliographicCitation.startpage88
local.contributor.affiliationStaines, H, College of Medicine, Biology and Environment, ANU
local.contributor.affiliationRae, Caroline, University of Sydney
local.contributor.affiliationKirk, Kiaran, College of Medicine, Biology and Environment, ANU
local.contributor.authoruidStaines, H, u931227
local.contributor.authoruidKirk, Kiaran, u9608579
local.description.embargo2037-12-31
local.description.notesImported from ARIES
local.description.refereedYes
local.identifier.absfor060110 - Receptors and Membrane Biology
local.identifier.absfor060502 - Infectious Agents
local.identifier.ariespublicationMigratedxPub18668
local.identifier.citationvolume1463
local.identifier.doi10.1016/S0005-2736(99)00187-X
local.identifier.scopusID2-s2.0-0033962141
local.type.statusPublished Version

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