Huynh, TonyUaesoontrachoon, KitipongQuinn, JamesTatem, Kathleen SHeier, ChristopherVan Der Meulen, Jack HYu, QingHarris, MarkNolan, ChristopherHaegeman, GuyGrounds, Miranda DNagaraju, Kanneboyina2015-12-100022-3417http://hdl.handle.net/1885/56027The over-expression of NF-κB signalling in both muscle and immune cells contribute to the pathology in dystrophic muscle. The anti-inflammatory properties of glucocorticoids, mediated predominantly through monomeric glucocorticoid receptor inhibition ofKeywords: atrogin 1; cathepsin B; ccl2 protein; creatine kinase; dystrophin; gamma interferon; glucocorticoid receptor; igf1 protein; immunoglobulin enhancer binding protein; interleukin 12p70; interleukin 6; mstn protein; osteopontin; prednisolone; protein; transc compound A; Duchenne muscular dystrophy; glucocorticoid receptor; glucocorticoids; transrepression NF- ? B201310.1002/path.42312016-02-24