Larter, ClaireYeh, MatthewHaigh, W GeoffreyVan Rooyen, DerrickBrooling, JohnHeydet, DeborahNolan, ChristopherTeoh, NarcissusFarrell, Geoffrey2015-12-131930-7381http://hdl.handle.net/1885/77691Background: Alms1 mutant (foz/foz) mice develop hyperphagic obesity, diabetes, metabolic syndrome, and fatty liver (steatosis). High-fat (HF) feeding converts pathology from bland steatosis to nonalcoholic steatohepatitis (NASH) with fibrosis, which leadsKeywords: adenosine triphosphate; alanine aminotransferase; CD36 antigen; cholesterol ester; fatty acid; immunoglobulin enhancer binding protein; triacylglycerol; uncoupling protein 2; adipose tissue; alanine aminotransferase blood level; animal cell; animal experi201310.1002/oby.201232016-02-24