Teoh, NarcissusFarrell, Geoff C2015-12-100815-9319http://hdl.handle.net/1885/51310This review highlights recent advances in our understanding of mechanisms underlying repeffusion injury to the liver after warm hepatic ischemia. Sinusoidal endothelial cells and hepatocytes are targets of injury in the early 'cytotoxic' phase, although pKeywords: cell adhesion molecule; chemokine; cytokine; interleukin 1; interleukin 6; nitric oxide; reactive oxygen metabolite; tumor necrosis factor; tumor necrosis factor alpha; cell death; energy metabolism; human; human cell; hypoxia; inflammatory cell; Kupffer Adenosine; Adhesion molecules; Cell-cycle entry; Hepatic ischemia reperfusion injury; Ischemic preconditioning; Kupffer cells; Microcirculation; Neutrophils; Nitric oxide; Reactive oxygen species; Tumor necrosis factor200310.1046/j.1440-1746.2003.03056.x2015-12-09