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Selective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx mice

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Date

2013

Authors

Huynh, Tony
Uaesoontrachoon, Kitipong
Quinn, James
Tatem, Kathleen S
Heier, Christopher
Van Der Meulen, Jack H
Yu, Qing
Harris, Mark
Nolan, Christopher
Haegeman, Guy

Journal Title

Journal ISSN

Volume Title

Publisher

John Wiley & Sons Inc

Abstract

The over-expression of NF-κB signalling in both muscle and immune cells contribute to the pathology in dystrophic muscle. The anti-inflammatory properties of glucocorticoids, mediated predominantly through monomeric glucocorticoid receptor inhibition of

Description

Keywords

Keywords: atrogin 1; cathepsin B; ccl2 protein; creatine kinase; dystrophin; gamma interferon; glucocorticoid receptor; igf1 protein; immunoglobulin enhancer binding protein; interleukin 12p70; interleukin 6; mstn protein; osteopontin; prednisolone; protein; transc compound A; Duchenne muscular dystrophy; glucocorticoid receptor; glucocorticoids; transrepression NF- ? B

Citation

URI

http://hdl.handle.net/1885/56027

Collections

ANU Research Publications

Source

Journal of Pathology

Type

Journal article

Book Title

Entity type

Access Statement

License Rights

DOI

10.1002/path.4231

Restricted until

2037-12-31

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Description
01_Huynh_Selective_modulation_through_2013.pdf (1.11 MB)

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