Mechanisms of Amyloid β Protein-Induced Modification in Ion Transport Systems: Implications for Neurodegenerative Diseases

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dc.contributor.authorKourie, Joseph
dc.date.accessioned2015-12-10T23:19:57Z
dc.date.issued2001
dc.date.updated2015-12-10T10:19:20Z
dc.description.abstract1. Alzheimer's disease (AD) is a neurodegenerative disorder that affects the cognitive function of the brain. Pathological changes in AD are characterized by the formation of amyloid plaques and neurofibrillary tangles as well as extensive neuronal loss.
dc.identifier.issn0272-4340
dc.identifier.urihttp://hdl.handle.net/1885/66127
dc.publisherKluwer Academic Publishers
dc.sourceCellular and Molecular Neurobiology
dc.subjectKeywords: adenosine triphosphatase (potassium sodium); adenosine triphosphate; amyloid beta protein; calcium channel; calcium ion; chloride channel; chloride ion; potassium channel; potassium ion; reactive oxygen metabolite; sodium channel; sodium ion; Alzheimer di Alzheimer's disease; Beta amyloid; Ion channel diseases; Membrane physiology; Neurodegeneration; Neurofibrillary tangles; Protein aggregation; Senile plaques
dc.titleMechanisms of Amyloid β Protein-Induced Modification in Ion Transport Systems: Implications for Neurodegenerative Diseases
dc.typeJournal article
local.bibliographicCitation.issue3
local.bibliographicCitation.lastpage213
local.bibliographicCitation.startpage173
local.contributor.affiliationKourie, Joseph, College of Physical and Mathematical Sciences, ANU
local.contributor.authoruidKourie, Joseph, u9111360
local.description.embargo2037-12-31
local.description.notesImported from ARIES
local.description.refereedYes
local.identifier.absfor110106 - Medical Biochemistry: Proteins and Peptides (incl. Medical Proteomics)
local.identifier.ariespublicationMigratedxPub1225
local.identifier.citationvolume21
local.identifier.doi10.1023/A:1010932603406
local.identifier.scopusID2-s2.0-0034858202
local.type.statusPublished Version

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