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The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: Role of endogenous TLR ligands

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Date

2013

Authors

Uaesoontrachoon, Kitipong
Cha, Hee-Jae
Ampong, Beryl
Sali, A
Vandermeulen, J
Wei, B
Creeden, B
Huynh, Tony
Quinn, James
Tatem, Kathleen S

Journal Title

Journal ISSN

Volume Title

Publisher

John Wiley & Sons Inc

Abstract

An absence of dysferlin leads to activation of innate immune receptors such as Toll-like receptors (TLRs) and skeletal muscle inflammation. Myeloid differentiation primary response gene 88 (MyD88) is a key mediator of TLR-dependent innate immune signallin

Description

Keywords

Keywords: dysferlin; myeloid differentiation factor 88; osteopontin; single stranded RNA; toll like receptor; toll like receptor 1; toll like receptor 13; toll like receptor 2; toll like receptor 4; toll like receptor 5; toll like receptor 6; toll like receptor 7; degeneration; dysferlin; inflammation; osteopontin; regeneration; single-stranded RNA; Toll-like receptor

Citation

URI

http://hdl.handle.net/1885/64027

Collections

ANU Research Publications

Source

Journal of Pathology

Type

Journal article

Book Title

Entity type

Access Statement

License Rights

DOI

10.1002/path.4207

Restricted until

2037-12-31

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01_Uaesoontrachoon_The_effects_of_MyD88_2013.pdf (922.59 KB)

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