The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: Role of endogenous TLR ligands
Date
2013
Authors
Uaesoontrachoon, Kitipong
Cha, Hee-Jae
Ampong, Beryl
Sali, A
Vandermeulen, J
Wei, B
Creeden, B
Huynh, Tony
Quinn, James
Tatem, Kathleen S
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John Wiley & Sons Inc
Abstract
An absence of dysferlin leads to activation of innate immune receptors such as Toll-like receptors (TLRs) and skeletal muscle inflammation. Myeloid differentiation primary response gene 88 (MyD88) is a key mediator of TLR-dependent innate immune signallin
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Keywords
Keywords: dysferlin; myeloid differentiation factor 88; osteopontin; single stranded RNA; toll like receptor; toll like receptor 1; toll like receptor 13; toll like receptor 2; toll like receptor 4; toll like receptor 5; toll like receptor 6; toll like receptor 7; degeneration; dysferlin; inflammation; osteopontin; regeneration; single-stranded RNA; Toll-like receptor
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Source
Journal of Pathology
Type
Journal article
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Restricted until
2037-12-31
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