Evaluation of interventions to reduce air pollution from biomass smoke on mortality in Launceston, Australia: retrospective analysis of daily mortality, 1994-2007

dc.contributor.authorJohnston, F. H.
dc.contributor.authorHanigan, Ivan
dc.contributor.authorHenderson, S. B.
dc.contributor.authorMorgan, G. G.
dc.date.accessioned2016-01-14T00:20:26Z
dc.date.available2016-01-14T00:20:26Z
dc.date.issued2013-01-08
dc.date.updated2016-02-24T08:56:45Z
dc.description.abstractOBJECTIVE: To assess the effect of reductions in air pollution from biomass smoke on daily mortality. DESIGN: Age stratified time series analysis of daily mortality with Poisson regression models adjusted for the effects of temperature, humidity, day of week, respiratory epidemics, and secular mortality trends, applied to an intervention and control community. SETTING: Central Launceston, Australia, a town in which coordinated strategies were implemented to reduce pollution from wood smoke and central Hobart, a comparable city in which there were no specific air quality interventions. PARTICIPANTS: 67 000 residents of central Launceston and 148 000 residents of central Hobart (at 2001 census). INTERVENTIONS: Community education campaigns, enforcement of environmental regulations, and a wood heater replacement programme to reduce ambient pollution from residential wood stoves started in the winter of 2001. MAIN OUTCOME MEASURES:Changes in daily all cause, cardiovascular, and respiratory mortality during the 6.5 year periods before and after June 2001 in Launceston and Hobart. RESULTS: Mean daily wintertime concentration of PM10 (particulate matter with particle size <10 µm diameter) fell from 44 µg/m³ during 1994-2000 to 27 µg/m³ during 2001-07 in Launceston. The period of improved air quality was associated with small non-significant reductions in annual mortality. In males the observed reductions in annual mortality were larger and significant for all cause (−11.4%, 95% confidence interval −19.2% to −2.9%; P=0.01), cardiovascular (−17.9%, −30.6% to −2.8%; P=0.02), and respiratory (−22.8%, −40.6% to 0.3%; P=0.05) mortality. In wintertime reductions in cardiovascular (−19.6%, −36.3% to 1.5%; P=0.06) and respiratory (−27.9%, −49.5% to 3.1%; P=0.07) mortality were of borderline significance (males and females combined). There were no significant changes in mortality in the control city of Hobart. CONCLUSIONS: Decreased air pollution from ambient biomass smoke was associated with reduced annual mortality in males and with reduced cardiovascular and respiratory mortality during winter months.
dc.description.sponsorshipThis study was supported by the Australian Research Council (LP0882048), the Tasmanian Government Department of Health and Human Services, and the Environment Protection Authority Tasmania.en_AU
dc.identifier.issn1756-1833en_AU
dc.identifier.urihttp://hdl.handle.net/1885/95377
dc.publisherBMJ Publishing Group
dc.relationhttp://purl.org/au-research/grants/arc/LP0882048
dc.rightsThis is an open-access article distributed under the terms of the Creative Commons Attribution Non-commercial License, which permits use, distribution, and reproduction in any medium, provided the original work is properly cited, the use is non commercial and is otherwise in compliance with the license. See: http://creativecommons.org/licenses/bync/2.0/ and http://creativecommons.org/licenses/by-nc/2.0/legalcode.
dc.sourceBMJ
dc.subjectKeywords: air pollution; air quality; article; Australia; cardiovascular mortality; female; human; humidity; male; mortality; particle size; particulate matter; pollution monitoring; priority journal; retrospective study; smoke; temperature; time series analysis; w
dc.titleEvaluation of interventions to reduce air pollution from biomass smoke on mortality in Launceston, Australia: retrospective analysis of daily mortality, 1994-2007
dc.typeJournal article
dcterms.dateAccepted2012-12-03
local.bibliographicCitation.issuejan08 12en_AU
local.bibliographicCitation.lastpagee8446en_AU
local.bibliographicCitation.startpagee8446en_AU
local.contributor.affiliationJohnston, Fay, University of Tasmania, Australiaen_AU
local.contributor.affiliationHanigan, Ivan, College of Medicine, Biology and Environment, CMBE Research School of Population Health, Natl Centre for Epidemiology & Population Health, The Australian National Universityen_AU
local.contributor.affiliationHenderson, Sarah, University of Tasmania, Australiaen_AU
local.contributor.affiliationMorgan, Geoffrey, University of Sydney, Australiaen_AU
local.contributor.authoruidu3171954en_AU
local.description.notesImported from ARIESen_AU
local.identifier.absfor111700en_AU
local.identifier.absfor111706en_AU
local.identifier.ariespublicationf5625xPUB2328en_AU
local.identifier.citationvolume346en_AU
local.identifier.doi10.1136/bmj.e8446en_AU
local.identifier.scopusID2-s2.0-84872292497
local.identifier.thomsonID000313553800018
local.publisher.urlhttp://www.bmj.com/company/en_AU
local.type.statusPublished Versionen_AU

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