Autophagy-independent functions of UVRAG are essential for peripheral naive T-cell homeostasis

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dc.contributor.authorAfzal, Samia
dc.contributor.authorHao, Zhenyue
dc.contributor.authorItsumi, Momoe
dc.contributor.authorAbouelkheer, Yasser
dc.contributor.authorBrenner, Dirk
dc.contributor.authorGao, Yunfei
dc.contributor.authorWakeham, Andrew
dc.contributor.authorHong, Claire
dc.contributor.authorLi, Wanda Y
dc.contributor.authorSylvester, Jennifer
dc.contributor.authorGilani, SO
dc.contributor.authorBruestle, Anne
dc.contributor.authorHaight, J
dc.contributor.authorYou-Ten, A
dc.contributor.authorLin, GH
dc.contributor.authorInoue, Satoshi
dc.contributor.authorMak, Tak W
dc.date.accessioned2015-12-10T23:13:14Z
dc.date.issued2015
dc.date.updated2015-12-10T09:37:55Z
dc.description.abstractUV radiation resistance-associated gene (UVRAG) encodes a tumor suppressor with putative roles in autophagy, endocytic trafficking, and DNA damage repair but its in vivo role in T cells is unknown. Because conditional homozygous deletion of Uvrag in mice
dc.identifier.issn0027-8424
dc.identifier.urihttp://hdl.handle.net/1885/64334
dc.publisherNational Academy of Sciences (USA)
dc.rightsAuthor/s retain copyrighten_AU
dc.sourcePNAS - Proceedings of the National Academy of Sciences of the United States of America
dc.titleAutophagy-independent functions of UVRAG are essential for peripheral naive T-cell homeostasis
dc.typeJournal article
dcterms.accessRightsOpen Accessen_AU
local.bibliographicCitation.issue4
local.bibliographicCitation.lastpage1124
local.bibliographicCitation.startpage1119
local.contributor.affiliationAfzal, Samia, University of Toronto
local.contributor.affiliationHao, Zhenyue, Ontario Cancer Institute
local.contributor.affiliationItsumi, Momoe, University of Health Network
local.contributor.affiliationAbouelkheer, Yasser, University Health Network
local.contributor.affiliationBrenner, Dirk, Ontario Cancer Institute
local.contributor.affiliationGao, Yunfei, University Health Network
local.contributor.affiliationWakeham, Andrew, Ontario Cancer Institute
local.contributor.affiliationHong, Claire, University Health Network
local.contributor.affiliationLi, Wanda Y, Ontario Cancer Institute
local.contributor.affiliationSylvester, Jennifer, University Health Network
local.contributor.affiliationGilani, SO, Ontario Cancer Institute
local.contributor.affiliationBruestle, Anne, College of Medicine, Biology and Environment, ANU
local.contributor.affiliationHaight, J, Ontario Cancer Institute
local.contributor.affiliationYou-Ten, A, Ontario Cancer Institute
local.contributor.affiliationLin, GH, Ontario Cancer Institute
local.contributor.affiliationInoue, Satoshi, Ontario Cancer Institute
local.contributor.affiliationMak, Tak W, University Health Network, Toronto
local.contributor.authoremailu5691124@anu.edu.au
local.contributor.authoruidBruestle, Anne, u5691124
local.description.notesImported from ARIES
local.identifier.absfor110703 - Autoimmunity
local.identifier.absseo920108 - Immune System and Allergy
local.identifier.ariespublicationa383154xPUB919
local.identifier.citationvolume112
local.identifier.doi10.1073/pnas.1423588112
local.identifier.scopusID2-s2.0-84921809383
local.identifier.uidSubmittedBya383154
local.type.statusPublished Version

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