Cell-mediated cytotoxicity in recovery from poxvirus infections

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dc.contributor.authorMullbacher, Arno
dc.date.accessioned2015-12-13T23:13:52Z
dc.date.available2015-12-13T23:13:52Z
dc.date.issued2003
dc.date.updated2015-12-12T08:36:01Z
dc.description.abstractThe availability of mutant and gene targeted knockout mice with defects in components of cellular cytotoxicity mediated by either the Fas or the exocytosis pathway permitted an analysis of their role in recovery from poxvirus infections. Ectromelia (EV),
dc.identifier.issn1052-9276
dc.identifier.urihttp://hdl.handle.net/1885/88328
dc.publisherJohn Wiley & Sons Inc
dc.sourceReviews in Medical Virology
dc.subjectKeywords: caspase; caspase 8; CD4 antigen; CD8 antigen; chromium 51; cysteine proteinase; DNA; Fas antigen; FAS ligand; gamma interferon; granzyme A; granzyme B; interleukin 18; iodine 125; lymphokine; major histocompatibility antigen class 1; perforin; proteinase
dc.titleCell-mediated cytotoxicity in recovery from poxvirus infections
dc.typeJournal article
local.bibliographicCitation.lastpage232
local.bibliographicCitation.startpage223
local.contributor.affiliationMullbacher, Arno, College of Medicine, Biology and Environment, ANU
local.contributor.authoremailu8102295@anu.edu.au
local.contributor.authoruidMullbacher, Arno, u8102295
local.description.notesImported from ARIES
local.description.refereedYes
local.identifier.absfor110799 - Immunology not elsewhere classified
local.identifier.ariespublicationMigratedxPub17982
local.identifier.citationvolume13
local.identifier.doi10.1002/rmv.381
local.identifier.scopusID2-s2.0-0038780024
local.identifier.uidSubmittedByMigrated
local.type.statusPublished Version

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