IL-5 contributes to worm expulsion and muscle hypercontractility in a primary T. spiralis infection
Enteric nematode infections lead to increased interleukin (IL)-5 expression, eosinophilic inflammation, and intestinal smooth muscle hypercontractility. Although eosinophils release inflammatory mediators that cause smooth muscle contraction, the role of
|Collections||ANU Research Publications|
|Source:||American Journal of Physiology: Gastrointestinal and Liver Physiology|
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