The Role of Endoplasmic Reticulum Stress in Nonimmune Diabetes: NOD.k iHEL, a Novel Model of β Cell Death
The final common pathway in diabetes development is β cell apoptosis. We herein describe a novel diabetes model based on transgenic NOD.k iHEL mice, wherein male mice develop diabetes due to nonimmune-mediated β cell death. Histology and electron micros
|Collections||ANU Research Publications|
|Source:||Annals of the New York Academy of Sciences|
|01_Socha_The_Role_of_Endoplasmic_2003.pdf||2.01 MB||Adobe PDF||Request a copy|
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