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The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: Role of endogenous TLR ligands

Uaesoontrachoon, Kitipong; Cha, Hee-Jae; Ampong, Beryl; Sali, A; Vandermeulen, J; Wei, B; Creeden, B; Huynh, Tony; Quinn, James; Tatem, Kathleen S; Rayavarapu, S


An absence of dysferlin leads to activation of innate immune receptors such as Toll-like receptors (TLRs) and skeletal muscle inflammation. Myeloid differentiation primary response gene 88 (MyD88) is a key mediator of TLR-dependent innate immune signallin

CollectionsANU Research Publications
Date published: 2013
Type: Journal article
Source: Journal of Pathology
DOI: 10.1002/path.4207


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