The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: Role of endogenous TLR ligands
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Uaesoontrachoon, Kitipong; Cha, Hee-Jae; Ampong, Beryl; Sali, A; Vandermeulen, J; Wei, B; Creeden, B; Huynh, Tony; Quinn, James; Tatem, Kathleen S; Rayavarapu, S
Description
An absence of dysferlin leads to activation of innate immune receptors such as Toll-like receptors (TLRs) and skeletal muscle inflammation. Myeloid differentiation primary response gene 88 (MyD88) is a key mediator of TLR-dependent innate immune signallin
| Collections | ANU Research Publications |
|---|---|
| Date published: | 2013 |
| Type: | Journal article |
| URI: | http://hdl.handle.net/1885/64027 |
| Source: | Journal of Pathology |
| DOI: | 10.1002/path.4207 |
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| File | Description | Size | Format | Image |
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| 01_Uaesoontrachoon_The_effects_of_MyD88_2013.pdf | 922.59 kB | Adobe PDF |  |
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