Novel folding and stability defects cause a deficiency of human glutathione transferase omega 1
The polymorphic deletion of Glu-155 from human glutathione transferase omega1 (GSTO1-1) occurs in most populations. Although the recombinant ΔGlu-155 enzyme expressed in Escherichia coli is active, the deletion causes a deficiency of the active enzyme in
|Collections||ANU Research Publications|
|Source:||Journal of Biological Chemistry|
|01_Zhou_Novel_folding_and_stability_2011.pdf||1.95 MB||Adobe PDF|
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