Infarct-induced chronic heart failure increases bi-directional protein movement across the alveolocapillary barrier
Chronic heart failure (CHF) is associated with adaptive structural changes at the alveolocapillary barrier that may be associated with altered protein permeability. Bidirectional protein movement across the barrier was studied in anesthetized rats with infarct-induced CHF by following 125I-labeled albumin (125I-albumin) flux into the alveoli and the leakage of surfactant protein (SP)-B from the alveoli into the circulation. Three groups were studied: controls [0% left ventricular (LV)...[Show more]
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|Source:||American Journal of Physiology: Heart and Circulatory Physiology|
|01_De Pasquale_Infarct-induced_chronic_heart_2003.pdf||329.44 kB||Adobe PDF||Request a copy|
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